Here’s early evidence that activity levels of HDL-associated antioxidant enzymes might prove to be a much-needed predictor of cardiovascular risk in systemic rheumatic diseases.
New, seemingly conflicting evidence on diet and heart health may make patients skeptical of any dietary guidance. Set them straight with this infographic on where the science stands.
When this CME summit was last offered two years ago, it drew hundreds of attendees from over 30 states and nine countries. Even more are expected this time. Here’s why you should be among them.
When dietary choline and L-carnitine meet gut flora, the result is TMAO — and much unhealthful mischief. New research shows the damage goes beyond atherosclerosis to heart failure, chronic kidney disease and maybe more.
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Further research revealed heart failure patients with elevated plasma levels of TMAO, a metabolite generated in the gut, have a higher mortality risk than patients with lower TMAO levels.
Researchers have found that a biochemical mechanism is at work between gut microbes and cholesterol that can lead to an increased risk of heart disease. They are looking more closely at a meat based diet vs. a plant based diet. Building on years of previous work, Dr. Hazen Stanley Hazen, Cleveland Clinic, and a research team are tracking the substance TMAO produced by the liver that is found to have an impact on negative cardiac outcomes.
Dr. Stanley Hazen’s long-term studies of HDL are profoundly altering our view of HDL and its role in heart disease — and may soon yield a diagnostic assay for cardiac risk based on the new insights.