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Probing Molecular Mechanisms of Epilepsy Progression

Recent Cleveland Clinic research shows increased expression of growth associated protein 43 (GAP-43) in dysplastic neurons, and an association between increased GAP-43 expression and longer epilepsy duration in patients with type II focal cortical dysplasia. These early findings suggest that GAP-43 may prove to be a pathology-specific biomarker for epileptogenicity and progression.